Road Rage

Road rage is an aggressive or angry behavior by a driver of an automobile or other motor vehicle. Such behavior might include rude gestures, verbal insults, deliberately driving in an unsafe or threatening manner, or making threats. Road rage can lead to altercations, assaults, and collisions which result in injuries and even deaths. It can be thought of as an extreme case of aggressive driving.
The term originated in the United States during the 1980s, specifically from Newscasters at KTLA, a local television station in Los Angeles, California. The term originated in 1987-1988, when a rash of freeway shootings occurred on the 405, 110 and 10 freeways in Los Angeles. These shooting sprees even spawned a response from the AAA Motor Club to its members on how to respond to drivers with road rage or aggressive maneuvers and gestures.
As early as 1997, therapists in the United States were working to certify road rage as a medical condition. It is not an official mental disorder in the Diagnostic and Statistical Manual of Mental Disorders. According to an article published by the Associated Press in June 2006, the behaviors typically associated with road rage are the result of intermittent explosive disorder. This conclusion was drawn from surveys of some 9,200 adults in the United States between 2001 and 2003 and was funded by the National Institute of Mental Health. The cause of intermittent explosive disorder has not been described to date. There are differing views on whether or not "road rage" is a mental issue.
A 2007 study of the largest U.S. metropolitan areas concluded that the cities with the least courteous drivers (most road rage) are Miami, Phoenix, New York, Los Angeles, and Boston. The cities with the most courteous drivers (least road rage) are Minneapolis, Nashville, St. Louis, Seattle, and Atlanta.
In spite of this, in 2009, New York, Dallas/Fort Worth, Detroit, Atlanta and Minneapolis/St. Paul were rated the top five "Road Rage Capitals" of the United States.

Water Intoxication

Water intoxication, also known as hyper-hydration, water poisoning, or overhydration, is a potentially fatal disturbance in brain functions that results when the normal balance of electrolytes in the body is pushed outside of safe limits by over-consumption of water.

Normal, healthy (physically, nutritionally and mentally) individuals have little reason to worry about accidentally consuming too much water. Nearly all deaths related to water intoxication in normal individuals have resulted either from water drinking contests, in which individuals attempt to consume large amounts of water, or long bouts of intensive exercise during which electrolytes are not properly replenished, yet excessive amounts of fluid are still consumed.

Water can be considered a poison when over-consumed just like any other substance. The recommendation from the medical field is to drink at least 1 - 2 liters per day depending upon body mass. Water intoxication would only occur at levels far higher than that.

Panama disease

Panama disease, a Fusarium wilt, is a banana plant disease caused by the fungus Fusarium oxysporum. The fungus attacks the roots of the banana plant. The disease is resistant to fungicide and cannot be controlled chemically.

Gros Michel or 'Big Mike' was an early export cultivar of banana. This cultivar was wiped out by Panama disease in the 1950s. The disease first appeared in Suriname, then made its way to the Caribbean, and, by the 1920s, to Honduras, the world's largest producer of bananas at the time. Although there are many banana cultivars, Gros Michel was especially suitable for export to non-tropical nations. A search for a substitute located the Vietnamese Cavendish cultivar, which is resistant to the disease. However, more care is required for shipping the Cavendish banana, and its quality compared to Gros Michel is debated.

Recently, a new strain called 'tropical race four Panama disease' has begun to attack Cavendish banana plants in south Asia. Given the high volume of modern international trade, banana producers expect this strain to spread through Africa and into South America and the Caribbean.

Plant breeders and geneticists are trying to develop new cultivars that are resistant to this new strain of Panama disease. Unfortunately, such efforts are progressing slowly because the triploid banana cultivars selected for human consumption are seedless and reproduce asexually, which decreases genetic variation and makes breeding difficult.

Münchausen Syndrome

Münchausen syndrome is a term for psychiatric disorders known as factitious disorders wherein those affected feign disease, illness, or psychological trauma in order to draw attention or sympathy to themselves. It is also sometimes known as hospital addiction syndrome or hospital hopper syndrome. Nurses sometimes refer to them as frequent flyers, because they return to the hospital just like frequent flyers return to the airport. However, there is discussion to reclassify them as somatoform disorder in the DSM-5 as it is unclear whether or not people are conscious of drawing attention to themselves.

In Münchausen syndrome, the affected person exaggerates or creates symptoms of illnesses in themselves to gain investigation, treatment, attention, sympathy, and comfort from medical personnel. In some extreme cases, people suffering from Münchausen's syndrome are highly knowledgeable about the practice of medicine and are able to produce symptoms that result in lengthy and costly medical analysis, prolonged hospital stay and unnecessary operations. The role of "patient" is a familiar and comforting one, and it fills a psychological need in people with Münchausen's. Risk factors for developing Münchausen syndrome include childhood traumas, and growing up with caretakers who, through illness or emotional problems, were unavailable.

The syndrome name derives from Baron Münchhausen (Karl Friedrich Hieronymus Freiherr von Münchhausen, 1720–1797) who purportedly told many fantastic and impossible adventures about himself, which Rudolf Raspe later published as The Surprising Adventures of Baron Münchhausen.

In 1951, Richard Asher was the first to describe a pattern of self-harm, where individuals fabricated histories, signs, and symptoms of illness. Remembering Baron Münchhausen, Asher named this condition Münchausen's Syndrome in his article in The Lancet in February 1951.

Paris Syndrome

Paris syndrome is a transient psychological disorder encountered by some people visiting or vacationing in Paris. It is similar in nature to Jerusalem syndrome and Stendhal syndrome.

Japanese visitors are observed to be especially susceptible. From the estimated six million yearly visitors the number of reported cases is significant: according to an administrator at the Japanese embassy in France, around twenty Japanese tourists a year are affected by the syndrome. The susceptilibity of Japanese people may be linked to the popularity of Paris in Japanese culture, notably the idealized image of Paris prevalent in Japanese media, which does not correspond to reality.

Paris Syndrome is characterized by a number of psychiatric symptoms such as acute delusional states, hallucinations, feelings of persecution (delusions of being a victim of prejudice, aggression, or hostility from others), derealization, depersonalization, anxiety, and also psychosomatic manifestations such as dizziness, tachycardia, sweating, etc.

In fact, the observed clinical picture is quite variable, but it has the characteristic of occurring during trips which confront travellers with things they have not previously experienced and had not anticipated. Principal to the diagnosis is that the experienced symptoms did not exist before the trip and disappear following a return to the sufferer's familiar surroundings.

Professor Hiroaki Ota, a Japanese psychiatrist working in France, is credited as the first person to diagnose the disease in 1986.

Tourette syndrome

Tourette syndrome is an inherited neuropsychiatric disorder with onset in childhood, characterized by multiple physical (motor) tics and at least one vocal (phonic) tic; these tics characteristically wax and wane. Tourette's is defined as part of a spectrum of tic disorders, which includes transient and chronic tics.

Tourette's was once considered a rare and bizarre syndrome, most often associated with the exclamation of obscene words or socially inappropriate and derogatory remarks (coprolalia), but this symptom is present in only a small minority of people with Tourette's.^[1] Tourette's is no longer considered a rare condition, but it may not always be correctly identified because most cases are classified as mild. Between 1 and 10 children per 1,000 have Tourette's;^[2] as many as 10 per 1,000 people may have tic disorders,^[3][4] with the more common tics of eye blinking, coughing, throat clearing, sniffing, and facial movements. People with Tourette's have normal life expectancy and intelligence. The severity of the tics decreases for most children as they pass through adolescence, and extreme Tourette's in adulthood is a rarity. Notable individuals with Tourette's are found in all walks of life.^[5]

Genetic and environmental factors play a role in the etiology of Tourette's, but the exact causes are unknown. In most cases, medication is unnecessary. There is no effective medication for every case of tics, but there are medications and therapies that can help when their use is warranted. Explanation and reassurance alone are often sufficient treatment;^[6] education is an important part of any treatment plan.^[7]

The eponym was bestowed by Jean-Martin Charcot (1825–1893) on behalf of his resident, Georges Albert Édouard Brutus Gilles de la Tourette (1859–1904), a French physician and neurologist, who published an account of nine patients with Tourette's in 1885.

Pica

Pica is a medical disorder characterized by an appetite for substances largely non-nutritive (e.g. metal [coins, etc.], clay, coal, soil, feces, chalk, paper, soap, mucus, ash, gum, etc.) or an abnormal appetite for some things that may be considered foods, such as food ingredients (e.g. flour, raw potato, raw rice, starch, ice cubes, salt). In order for these actions to be considered pica, they must persist for more than one month at an age where eating such objects is considered developmentally inappropriate. The condition's name comes from the Latin word for magpie, a bird that is reputed to eat almost anything. Pica is seen in all ages, particularly in pregnant women, small children, and those with developmental disabilities.

Pica in children, while common (usually only in young children or children with autism or another mental or developmental disorder), may be dangerous. Children eating painted plaster containing lead may suffer brain damage from lead poisoning. There is a similar risk from eating dirt near roads that existed prior to the phaseout of tetra-ethyl lead in gasoline (in some countries) or prior to the cessation of the use of contaminated oil (either used, or containing toxic PCBs or dioxin) to settle dust. In addition to poisoning, there is also a much greater risk of gastro-intestinal obstruction or tearing in the stomach. This is also true in animals. Another risk of dirt-eating is the possible ingestion of animal feces and accompanying parasites. Pica can also be found in animals, and is most commonly found in dogs.

The scant research that has been done on the causes of pica suggests that the disorder is a specific appetite caused by mineral deficiency in many cases, in females named typically iron deficiency, which sometimes is a result of celiac disease. Often the substance eaten by someone with pica contains the mineral in which that individual is deficient. More recently, cases of pica have been tied to the obsessive–compulsive spectrum, and there is a move to consider OCD in the etiology of pica; however, pica is currently recognized as a mental disorder by the widely used Diagnostic and Statistical Manual of Mental Disorders (DSM-IV). Sensory, physiological, cultural, and psychosocial perspectives have also been used by some to explain the causation of pica.

Mental health conditions, such as obsessive-compulsive disorder (OCD) and schizophrenia can sometimes cause pica. It was suggested that stress associated with traumatic events is linked to pica disorder. Some of the traumatic events common in individuals with pica include maternal deprivation, parental separation or neglect, child abuse, disorganized family structure and poor parent-child interaction. Pica may also be a symptom of iron deficiency anemia secondary to hookworm infection.

However, pica can also be a cultural practice not associated with a deficiency or disorder. Ingestion of kaolin (white dirt) among African-American women in the U.S. state of Georgia shows the practice there to be a DSM-IV "culture-bound syndrome" and "not selectively associated with other psychopathology". Similar kaolin ingestion is also widespread in parts of Africa. Such practices might stem from health benefits such as clay's ability to absorb plant toxins and protect against toxic alkaloids and tannic acids.

Unlike in humans, pica in dogs or cats may be a sign of immune-mediated hemolytic anemia, especially when it involves eating substances such as tile grout, concrete dust, and sand. Dogs exhibiting this form of pica should be tested for anemia with a CBC or at least hematocrit levels.
ccompanying parasites. Pica can also be found in animals, and is most commonly found in dogs.

Lead Poisoning

Lead poisoning is a medical condition caused by increased levels of the heavy metal lead in the body. Lead interferes with a variety of body processes and is toxic to many organs and tissues including the heart, bones, intestines, kidneys, and reproductive and nervous systems. It interferes with the development of the nervous system and is therefore particularly toxic to children, causing potentially permanent learning and behavior disorders. Symptoms include abdominal pain, headache, anemia, irritability, and in severe cases seizures, coma, and death.

Routes of exposure to lead include contaminated air, water, soil, food, and consumer products. Occupational exposure is a common cause of lead poisoning in adults. One of the largest threats to children is lead paint that exists in many homes, especially older ones; thus children in older housing with chipping paint are at greater risk. Prevention of lead exposure can range from individual efforts (e.g. removing lead-containing items such as piping or blinds from the home) to nationwide policies (e.g. laws that ban lead in products or reduce allowable levels in water or soil).

Elevated lead in the body can be detected by the presence of changes in blood cells visible with a microscope and dense lines in the bones of children seen on X-ray. However, the main tool for diagnosis is measurement of the blood lead level; different treatments are used depending on this level. The major treatments are removal of the source of lead and chelation therapy (administration of agents that bind lead so it can be excreted).

No safe threshold for lead exposure has been discovered—that is, there is no known amount of lead that is too small to cause the body harm.

Lead poisoning was among the first known and most widely studied work and environmental hazards. One of the first metals to be smelted and used, lead is thought to have been discovered and first mined in Anatolia around 6500 BCE. Its density, workability, and corrosion-resistance were among the metal's attractions.

In the second century BCE the Greek botanist Nicander described the colic and paralysis seen in lead-poisoned people. Dioscorides, a Greek physician who lived in the first century CE, wrote that lead makes the mind "give way".

Lead was used extensively in Roman aqueducts from about 500 BCE to 300 CE Julius Caesar's engineer, Vitruvius, reported, "water is much more wholesome from earthenware pipes than from lead pipes. For it seems to be made injurious by lead, because white lead is produced by it, and this is said to be harmful to the human body." Gout, prevalent in affluent Rome, is thought to be the result of lead, or leaded eating and drinking vessels. Sugar of lead was used to sweeten wine, and the gout that resulted from this was known as saturnine gout.

After antiquity, mention of lead poisoning was absent from medical literature until the end of the Middle Ages. In 1656 the German physician Samuel Stockhausen recognized dust and fumes containing lead compounds as the cause of disease, called since ancient Roman times morbi metallici, that were known to afflict miners, smelter workers, potters, and others whose work exposed them to the metal.

The painter Caravaggio might have died of lead poisoning. Bones with high lead levels were recently found in a grave likely to be Caravaggio's grave. Paints used at the time contained high amounts of lead salts. Caravaggio is known to have indulged in violent behavior, as caused by lead poisoning.

In 17th-century Germany, the physician Eberhard Gockel discovered lead-contaminated wine to be the cause of an epidemic of colic. He had noticed that monks who did not drink wine were healthy, while wine drinkers developed colic, and traced the cause to sugar of lead, made by simmering litharge with vinegar. As a result, Eberhard Ludwig, Duke of Württemberg issued an edict in 1696 banning the adulteration of wines with litharge.

In 18th century Boston, lead poisoning was fairly frequent on account of the widespread drinking of rum, which was made in stills with a lead component (the "worm"). Also in the 18th century, "Devonshire colic" was the name given to the symptoms suffered by people of Devon who drank cider made in presses that were lined with lead. Lead was added to cheap wine illegally in the 18th and early 19th centuries as a sweetener. The composer Beethoven, a heavy wine drinker, suffered elevated lead levels (as later detected in his hair) possibly due to this; the cause of his death is controversial, but lead poisoning is a contender as a factor.

With the Industrial Revolution in the 19th century, lead poisoning became common in the work setting. The introduction of lead paint for residential use in the 19th century increased childhood exposure to lead; for millennia before this, most lead exposure had been occupational. An important step in the understanding of childhood lead poisoning occurred when toxicity in children from lead paint was recognized in Australia in 1897. France, Belgium and Austria banned white lead interior paints in 1909; the League of Nations followed suit in 1922. However, in the United States, laws banning lead house paint were not passed until 1971, and it was phased out and not fully banned until 1978.

The 20th century saw an increase in worldwide lead exposure levels due to the increased widespread use of the metal. Beginning in the 1920s, lead was added to gasoline to improve its combustion; lead from this exhaust persists today in soil and dust in buildings. Blood lead levels worldwide have been declining sharply since the 1980s, when leaded gasoline began to be phased out. In those countries that have banned lead in solder for food and drink cans and have banned leaded gasoline additives, blood lead levels have fallen sharply since the mid-1980s.

The levels found today in most people are orders of magnitude greater than those of pre-industrial society. Due to reductions of lead in products and the workplace, acute lead poisoning is rare in most countries today; however, low level lead exposure is still common. It was not until the second half of the 20th century that subclinical lead exposure became understood to be a problem. During the end of the 20th century, the blood lead levels deemed acceptable steadily declined. Blood lead levels once considered safe are now considered hazardous, with no known safe threshold.

Retinal Detachment

Retinal detachment is a disorder of the eye in which the retina peels away from its underlying layer of support tissue. Initial detachment may be localized, but without rapid treatment the entire retina may detach, leading to vision loss and blindness. It is a medical emergency.

The retina is a thin layer of light sensitive tissue on the back wall of the eye. The optical system of the eye focuses light on the retina much like light is focused on the film in a camera. The retina translates that focused image into neural impulses and sends them to the brain via the optic nerve. Occasionally, posterior vitreous detachment, injury or trauma to the eye or head may cause a small tear in the retina. The tear allows vitreous fluid to seep through it under the retina, and peel it away like a bubble in wallpaper.

• Rhegmatogenous retinal detachment – A rhegmatogenous retinal detachment occurs due to a break in the retina that allows fluid to pass from the vitreous space into the subretinal space between the sensory retina and the retinal pigment epithelium. Retinal breaks are divided into three types - holes, tears and dialyses. Holes form due to retinal atrophy especially within an area of lattice degeneration. Tears are due to vitreoretinal traction. Dialyses which are very peripheral and circumferential may be either tractional or atrophic, the atrophic form most often occurring as idiopathic dialysis of the young.
• Exudative, serous, or secondary retinal detachment – An exudative retinal detachment occurs due to inflammation, injury or vascular abnormalities that results in fluid accumulating underneath the retina without the presence of a hole, tear, or break. In evaluation of retinal detachment it is critical to exclude exudative detahment as surgery will make the situation worse not better. Although rare, exudative retinal detachment can be caused by the growth of a tumor on the layers of tissue beneath the retina, namely the choroid. This cancer is called a choroidal melanoma.
• Tractional retinal detachment – A tractional retinal detachment occurs when fibrous or fibrovascular tissue, caused by an injury, inflammation or neovascularization, pulls the sensory retina from the retinal pigment epithelium.

A minority of retinal detachments result from trauma, including blunt blows to the orbit, penetrating trauma, and concussions to the head. A retrospective Indian study of more than 500 cases of rhegmatogenous detachments found that 11% were due to trauma, and that gradual onset was the norm, with over 50% presenting more than one month after the inciting injury.

White Nose Syndrome

White nose syndrome is a poorly understood malady associated with the deaths of more than a million bats. The condition, named for a distinctive fungal growth around the muzzles and on the wings of many affected animals, was first identified in a cave in Schoharie County, New York, USA, in February 2006, and started showing up in the news after January 2007. It spread to other New York caves and into Vermont, Massachusetts and Connecticut in 2008. In early 2009 it was confirmed in New Hampshire, New Jersey, Pennsylvania, West Virginia and in March 2010 in Ontario, Canada, and northern Tennessee. As of spring 2010, the condition had been found in over 115 caves and mines ranging throughout the Northeastern US as far south as Tennessee and as far west as Oklahoma and into Quebec and Ontario Provinces in Canada .

The US Fish & Wildlife Service (USFWS) has called for a moratorium on caving activities in the affected areas, and strongly recommends that any clothing or equipment used in such areas be decontaminated after each use.

The National Speleological Society (NSS) maintains an up-to-date page to keep cavers apprised of current events and advisories.

Argyria

Argyria is a condition caused by improper exposure to chemical forms of the element silver, silver dust, or silver compounds. The most dramatic symptom of argyria is that the skin becomes blue or bluish-grey colored. Argyria may be found as generalized argyria or local argyria. Argyrosis is the corresponding condition related to the eye. The condition is believed to be permanent, but laser therapy has been used to treat it with satisfactory cosmetic results.

In animals and humans, silver accumulates in the body over time. Chronic intake of silver products can result in an accumulation of silver or silver sulfide particles in the skin. As in photography (where silver is used due to its reactivity with light), these particles in the skin darken with exposure to sunlight, resulting in a blue or gray discoloration of the skin. This condition is known as argyria. Chronic ingestion of silver can similarly lead to an accumulation of silver in the eye (argyrosis) and in other organs. Localized argyria can occur as a result of topical use of substances containing silver, while generalized argyria results from the chronic ingestion of such substances. Argyria is generally believed to be irreversible, with the only practical method of minimizing its cosmetic disfigurement being to avoid the sun, but laser therapy has been used to treat it with satisfactory cosmetic results. The Agency for Toxic Substances and Disease Registry (ATSDR) describes argyria as a "cosmetic problem", which is not harmful, but it is mildly disfiguring and thus some people find it to be socially debilitating.

Ice Cream Headache

An ice cream headache, also known as brain freeze, cold-stimulus headache, or its given scientific name sphenopalatine ganglioneuralgia (meaning "nerve pain of the sphenopalatine ganglion"), is a form of brief cranial pain or headache commonly associated with consumption (particularly quick consumption) of cold beverages or foods such as ice cream and popsicles. It is caused by having something cold touch the roof of the mouth (palate), and is believed to result from a nerve response causing rapid dilation and swelling of blood vessels or a "referring" of pain from the roof of the mouth to the head. The rate of intake for cold foods has been studied as a contributing factor.

"Ice cream headaches" result from quickly eating or drinking very cold substances. It is commonly experienced when applying ice-cream (or similar) to the roof of the mouth (palate) or when swallowing it. Typically the headache appears in about 10 seconds and lasts about 20 seconds although some people experience much longer lapses of pain, with the pain seeming to relate to the same side of the head as the cold substance was applied to the palate, or to both sides of the head in the case of swallowing. The most effective way to prevent it is to consume the cold food or liquid at a slower rate. Keeping it in one's mouth long enough for the palate to become used to the temperature is also an effective preventative.

Ice cream headache is the direct result of the rapid cooling and rewarming of the capillaries in the sinuses. A similar but painless blood vessel response causes the face to appear "flushed" after being outside on a cold day. In both instances, the cold temperature causes the capillaries in the sinuses to constrict and then experience extreme rebound dilation as they warm up again.

In the palate, this dilation is sensed by nearby pain receptors, which then send signals back to the brain via the trigeminal nerve, one of the major nerves of the facial area. This nerve also senses facial pain, so as the nerval signals are conducted the brain interprets the pain as coming from the forehead—the same "referred pain" phenomenon seen in heart attacks. Brain-freeze pain may last from a few seconds to a few minutes. Research suggests that the same vascular mechanism and nerve implicated in "brain freeze" cause the aura (sensory disturbance) and pulsatile (throbbing pain) phases of migraines.

It is possible to suffer from an ice-cream headache in both hot and cold climate conditions.

To relieve pain, some doctors suggest pressing the tongue against the roof of the mouth to warm the area.

The phenomenon is common enough to have been the subject of research published in the British Medical Journal and Scientific American.

Hangover

A hangover (veisalgia) describes the sum of unpleasant physiological effects following heavy consumption of alcoholic beverages. An alcohol hangover is associated with a variety of symptoms that may include dehydration, fatigue, headache, nausea, vomiting, diarrhea, flatulence, weakness, elevated body temperature, hypersalivation, difficulty concentrating, sweating, anxiety, dysphoria, irritability, sensitivity to light and noise, erratic motor functions (including tremor), trouble sleeping, severe hunger, halithosis, and lack of depth perception. Many people will also be repulsed by the thought, taste or smell of alcohol during a hangover. The symptoms vary from person to person, and occasion to occasion, usually beginning several hours after drinking. It is not clear whether hangovers directly affect cognitive abilities.

A hangover may also induce psychological symptoms including heightened feelings of depression and anxiety.

The term hangover was originally a 19th Century expression describing unfinished business—something left over from a meeting—or "survival". In 1904, the meaning "morning after-effect of drinking too much" first surfaced.

Hypoglycemia, dehydration, acetaldehyde intoxication, and vitamin B12 deficiency are all theorized causes of hangover symptoms. Hangovers symptoms may persist for several days after alcohol was last consumed. Approximately 25-30% of drinkers may be resistant to hangover symptoms. Some aspects of a hangover are viewed as symptoms of acute ethanol withdrawal, similar to the longer-duration effects of withdrawal from alcoholism, as determined by studying the increases in brain reward thresholds in rats (the amount of current required to receive to electrodes implanted in the lateral hypothalamus) following ethanol injection.

Decompression Sickness

Decompression sickness (DCS; also historically or colloquially known as divers' disease, the bends or caisson disease) describes a condition arising from the precipitation of dissolved gasses into bubbles inside the body on depressurisation. DCS most commonly refers to a specific type of scuba diving hazard but may be experienced in other depressurisation events such as caisson working, flying in unpressurised aircraft and extra-vehicular activity from spacecraft.

Although DCS is not a common event, its potential severity is such that much research has gone into preventing it and scuba divers use dive tables or dive computers to set limits to their exposure to pressure. Its effects may vary from joint pain and rash to paralysis and death. Treatment is by hyperbaric oxygen therapy in a recompression chamber. If treated early, there is a significantly higher chance of success.

The original name for DCS was caisson disease; this term was used in the 19th century, in large engineering excavations below the water table, such as with the piers of bridges and with tunnels, where caissons under pressure were used to keep water from flooding the excavations. Workers who spend time in high-pressure atmospheric pressure conditions are at risk when they return to the lower pressure outside the caisson without slowly reducing the surrounding pressure.

DCS was a major factor in the during construction of Eads Bridge, when 15 workers died from what was then a mysterious illness, and later during construction of the Brooklyn Bridge, where it incapacitated the project leader Washington Roebling.

Stockholm Syndrome

Stockholm syndrome is a psychological response sometimes seen in abducted hostages, in which the hostage shows signs of loyalty to the hostage-taker, regardless of the danger or risk in which they have been placed. The syndrome is named after the Norrmalmstorg robbery of Kreditbanken at Norrmalmstorg in Stockholm, in which the bank robbers held bank employees hostage from August 23 to August 28, 1972. In this case, the victims became emotionally attached to their captors, and even defended them after they were freed from their six-day ordeal. The term "Stockholm Syndrome" was coined by the criminologist and psychiatrist Nils Bejerot, who assisted the police during the robbery, and referred to the syndrome in a news broadcast.

The Stockholm Syndrome is a psychological shift that occurs in captives when they are threatened gravely but shown acts of kindness by their captors. Captives who exhibit the syndrome tend to sympathize with and think highly of their captors, at times believing that the captors are showing them favor stemming from inherent kindness. Such captives fail to recognize that their captors' choices are essentially self-serving. When subjected to prolonged captivity, these captives can develop a strong bond with their captors, in some cases including a sexual interest.

According to the psychoanalytic view of the syndrome, this tendency might be the result of employing the strategy evolved by newborn babies to form an emotional attachment to the nearest powerful adult in order to maximize the probability that this adult will enable — at the very least — the survival of the child, if not also prove to be a good parental figure. This syndrome is considered a prime example for the defense mechanism of identification.

The syndrome has also been explained in evolutionary terms, with reference to the fact that our ancestors sided with the tribes that captured them. In many cases, capture may also involve the killing of the captive's relatives, thereby isolating the captive. The captive is subjected to isolation and so sees even a small act, such as providing amenities, as a great favor. Such captives may side with their captors while believing their captors have conferred on them great importance and love. Furthermore, captives who perceive themselves as the only members of their group not to have been killed may believe that they have been shown a special interest.